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Cachexia is a metabolic syndrome that manifests with excessive weight loss and disproportionate muscle wasting. It is related to many different chronic diseases, such as cancer, infections, liver disease, inflammatory bowel disease, cardiac disease, chronic obstructive pulmonary disease, chronic renal failure and rheumatoid arthritis. Cachexia is linked with poor outcome for the patients. In this article, we explore the role of the hypothalamus, liver, muscle tissue and adipose tissue in the pathogenesis of this syndrome, particularly concentrating on the role of cytokines, hormones and cell energy-controlling pathways (such as AMPK, PI3K/Akt and mTOR). We also look at possible future directions for therapeutic strategies.

Original publication

DOI

10.1586/eem.11.87

Type

Journal article

Journal

Expert Rev Endocrinol Metab

Publication Date

01/2012

Volume

7

Pages

73 - 90

Keywords

adipose tissue, cachexia, hypothalamus, malnutrition, molecular mechanisms, muscle tissue, pathophysiology, sarcopenia, wasting